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Conference to look at reversing and preventing alcohol-induced dementia
A press conference will be held on the problem of alcohol dementia as part of the 21st ECNP Congress 2008 which will be held in Barcelona, Spain from 30th August to 3rd September 2008.
Description
After many years of heavy drinking, alcohol produces pathological alterations in the brain. In many alcoholics these changes culminate in disorders of memory and learning. Severe cognitive impairments occur in approximately 10% of heavy drinkers. Alcoholic dementia is the second leading cause of adult dementia in the Western countries, accounting for 10% of all cases, and still represents an unresolved problem. So far no effective pharmacotherapy for memory problems in alcoholics is available.
Nowadays this problem can be approached by innovative research using molecular and epigenetic analyses, which yield new insight into brain pathophysiology. Molecular dysregulations in endogenous opioids – a neurotransmitter system in the brain that is central to reward function and pain control – are supposed to play a critical role in the development of alcoholism and associated cognitive impairment.
Professor Georgy Bakalkin, involved in a research team focusing on biochemical mechanisms underlying neuropsychiatric disorders, will highlight the latest findings on this clinical challenge at a press conference as part of the 21st ECNP Congress 2008. An emerging concept that proposes a new target for pharmacotherapy will be discussed, as will possible innovative therapeutic interventions to improve or prevent alcohol-induced cognitive impairment in patients with alcohol dependence.
Professor Bakalkin will hold the press conference on 2nd September 2008.
Article published on 24/07/2008 by DryOutNow.com
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